CLEC4E and myocardial infarction: Mincle could communicate with another C‐type lectin receptor or toll like receptors by forming a heterodimer, which may expand ligand specificity and confer multiple functions.54 Therefore, our next study will primarily focus on the molecular pathway which induces Mincle expression and the connections between Mincle and other innate immune receptors in sympathetic hyperactivity after myocardial infarction.