CLN3 and Miyoshi myopathy: Recently, DNA aptamer CLN0003 (CLN3) was isolated from Jurkat cells via Cell‐ SELEX and was found to bind c‐met with high specificity and affinity.20 Ueki et al identified the 50‐mer minimal binding motif of CLN3 (SL1) that retained high c‐met affinity and interfered with HGF binding to c‐met in SNU‐5 cells.21 However, whether SL1 can become the first aptamer to target c‐met in MM requires further investigation.