HGF is frequently present in the BMME where it activates the c‐met receptor in MM cells in a paracrine or autocrine fashion.5 We evaluated the mRNA expression of c‐met in two independent publicly available data sets and showed that c‐met transcript levels gradually increased from healthy donors with low expression to MGUS patients with intermediate expression and then to MM patients with high expression. This evidence concerns the gene HGF and Miyoshi myopathy.