As a co‐receptor, CD138 strongly promotes HGF‐induced signaling by promoting the dimerization of c‐met.33 c‐met mRNA and protein expression were found to be higher in CD138+ cells than in CD138‐ cells.11 These results support the potential clinical use of SL1 by only targeting MM cells as opposed to healthy cells. This evidence concerns the gene HGF and Miyoshi myopathy.