He was originally normocalcemic but after the craniosynostosis surgery, he developed severe hypocalcemia, causing a circulatory shock and a disseminated intravascular coagulation that led to a lethal cerebral infarction.19 A second reported child did not have a lethal outcome but a severe form of pseudohypoparathyroidism that is associated with morbid obesity.20 He showed TSH and PTH resistance, diagnosed at the age of 3.6 years, and prothrombotic status. Here, PTH is linked to craniosynostosis.