IL-4, IL-5, and IL-10 are secretions of Th2 cells, whose overexpression can lead to an imbalance of proportion between Th1 and Th2 cells and promote the conversion of Th2 cells so as to aggravate the conditions.15-17 Moreover, the levels of IL-5 and IL-10, adhesion molecules, and sE-selectin in the combined group were significantly higher than those in the allergic rhinitis group and the asthma group (P<0.05). The gene discussed is IL5; the disease is asthma.