Indeed, previous studies have shown that the G1 and G2 risk variants of the APOL1 gene are in strong linkage disequilibrium with variants in MYH9. Indeed, most of the association previously attributed to MYH9 variants or haplotypes with CKD could be explained by their genetic linkage with APOL1 polymorphisms in populations of African ancestry residing outside the African continent [35, 36]. This evidence concerns the gene MYH9 and chronic kidney disease.