NGFR and melanoma: Trametinib‐induced CD271 signalling has recently been shown to induce prosurvival DNA damage response pathways,18 and data from the present study additionally suggest that such signalling may promote survival by inhibiting cytotoxic autophagy as knock‐down of trametinib‐induced CD271 in WM35 melanoma cells resulted in increased autophagy and reduced cell viability.