Moreover, application of antago‐miR‐17‐5p could impede growth of chemo‐resistant neuroblastoma by blocking cell cycle and accelerating cell apoptosis.41 For another, elevated miR‐17‐5p expression could raise lung cancer cells’ resistance to docetaxel via prohibiting expression of autophagy‐relevant proteins (eg Beclin l).42 Consistently, it was explained in this study that miR‐17‐5p was subject to regulation of HOTAIR in its increasing cell viability and decreasing cell apoptosis, thereby down‐regulating gastric cancer cells’ chemosensitivity (Figures 3, 4). Here, HOTAIR is linked to lung cancer.