This is in contrast to findings pertaining to KCNJ2-linked SQTS presented in our previous study (Whittaker et al., 2017b), where reduced WL was found to be mediated by a decrease in both ERP and CV; this emphasises the value of multi-scale computational modelling in elucidating phenotypic differences between different variants of the SQTS. The gene discussed is KCNJ2; the disease is Familial short QT syndrome.