Our functional enrichment of genes in patient tumors revealed that low expression of RND1 in glioblastoma induces an overexpression of focal adhesion proteins like extracellular matrix proteins (COL1A1 and LAMB1); integrins (ITGA5 and ITGB1); actin-binding proteins (FLNA; ACTN1) and vinculin (Supplementary Table 5). This evidence concerns the gene COL1A1 and glioblastoma.