When tested in mice, they markedly reduce the levels of TNF-α and IL-1β [14], as well as the anti-tumor virus in liver, lung, and breast cancer, by preventing tumor cell proliferation and inducing tumor cell differentiation [15,16,17,18], in addition to the degree of alcohol-induced steatosis; they also contribute to improving the immune capacity and potentially treat alcoholic liver disease (ALD) [19]. The gene discussed is TNF; the disease is neoplasm.