Third, numerous genetic association studies and investigations utilizing genetically modified mouse models supported an important role of SLC26A9 in the pathophysiology of several organ manifestations in CF, as well as other chronic lung diseases such as asthma and non-CF bronchiectasis (Anagnostopoulou et al., 2012; Sun et al., 2012; Bakouh et al., 2013; Blackman et al., 2013; Miller et al., 2015; Strug et al., 2016). The gene discussed is SLC26A9; the disease is bronchiectasis.