KCNK13 and Alzheimer disease: The impairment of motility of microglial processes that occurs in some pathological conditions, e.g., in models of Alzheimer's disease with Aβ plaque deposition (Koenigsknecht-Talboo et al., 2008; Krabbe et al., 2013; Condello et al., 2015) raises the question of whether the dependence of surveillance on THIK-1 activity can be employed therapeutically for the treatment of AD (Madry et al., 2018).