Furthermore, NA has been tested as an antimicrobial therapeutic agent in murine models of infection caused by opportunistic pathogens, including Staphylococcus aureus and Citrobacter rodentium, and was found to stimulate host neutrophil cells to release microbicidal peptides by direct activation of the myeloid-specific transcription factor CCAAT/enhancer-binding protein ε (C/EBPε) (13, 14). The gene discussed is CEBPE; the disease is infection.