For instance, miR let-7, which is aberrantly expressed in myocardial infarction, arrhythmia, angiogenesis, atherosclerosis, and hypertension57, has also been reported to inhibit Bcl-xl expression and to mediate ox-LDL-induced endothelial apoptosis through the regulation of cell proliferation, migration, autophagy and apoptosis58. Here, BCL2L1 is linked to myocardial infarction.