ULK1 and acute myeloid leukemia: AMPK and ULK1 were found to have a similar cytoprotective mechanism against chemotherapeutics in primary pancreatic cancer cells as well as pancreatic cell lines.192 Further, in a t(8;21) AML model, Kasumi‐1 cells survived short‐term treatment with histone deacetylase inhibitors by upregulation of autophagy.193 However, interactions between AMPK and mTOR were not investigated and long‐term resistance was not examined.