Based on the strain genotype (babA, sabA and labA status) and mucin glycosylation, a similar proportion of the strains are expected to bind to mucins among the PUD and NUD strains at pH 7: 8/11 PUD vs 6/9 NUD would theoretically be expected to bind to the mucin from the human gastric tumor mucosa (Leb+, SLex+, LacdiNAc+, sulfate+), 9/11 PUD vs 9/9 NUD to the mucin from the human gastric normal mucosa (Leb+, SLex-, LacdiNAc+) and 5/11 PUD vs 5/9 NUD to the mucin from the monkey normal gastric mucosa (Leb-, SLex+, LacdiNAc−, Table 3). The gene discussed is MUC5AC; the disease is gastric neoplasm.