The highly significant proteins were categorised into five groups depending on their functions and association with the pathogenesis of steatosis: (i) apoptosis and autophagy, (ii) energy maintenance, (iii) anti-oxidative stress, (iv) liver injury (e.g. fat deposition) and (v) liver function improvement (e.g. nitrogen waste detoxicfication and folate-binding protein). Here, FOLR1 is linked to steatosis.