Gene variants involving the production and metabolism of bilirubin are risk factors of neonatal jaundice – including glucose-6-phosphate dehydrogenase (G6PD)2, blood group3, heme oxygenase (HO)−14, hepatic solute carrier organic anion transporter 1B1 (SLCO1B1)5, and UDP-glucuronosyltransferase 1A1 (UGT1A1)6,7. Here, UGT1A1 is linked to neonatal jaundice.