Genome-wide association studies (GWAS) have shownd that HLA-DP (rs3117242) variants contribute to the pathogenesis of MPO-ANCA associated vasculitis, while PRTN3 (rs 62,132,295) variants might contribute to PR3-ANCA associated vasculitis [21, 34–37]. Here, PRTN3 is linked to anti-neutrophil cytoplasmic antibody-associated vasculitis.