Cytokines and soluble signalling molecules were more abundant in those with pneumonia, with the exception of macrophage inflammatory protein (MIP)-1beta and MIP-1alpha, whereas the oxylipins were more abundant in those with brain injuries, with the exception of tetranor-prostaglandin D metabolite (tetranor-PGDM), leukotriene C4 and lipoxin B4 (Fig. 1). Here, CCL4L2 is linked to pneumonia.