For HF/n-3 mice, our data indicated that the supply of FFA for mitochondria seems more likely be sustained by increased lipid uptake (LPL and CD36) than lipolysis of TAG, but they also indicated subsequent efficient fatty acid oxidation (CPT1a, PPARα, FABP3 [51]), and ultimately enhanced lipid turnover, as well as a balanced energy metabolism (Table S6B). This evidence concerns the gene CPT1A and hydrops fetalis.