CAMP and psoriasis: In these patients, UVB radiation may induce innate immunity through antimicrobial peptides such as cathelicidin LL‐37, resulting in psoriatic lesions when there is simultaneous resistance to the UV‐induced suppression of the adaptive immune response (like in PLE) that would normally counteract such lesions.9 Thus, patients with photosensitive psoriasis may not be allocated to UVB phototherapy (based on a history of photoaggravation that is possibly linked to PLE), which may in turn explain the very low prevalence of PLE in our series of psoriasis patients.