PSMC5 and bacterial infectious disease: S3A and S3B Fig shows that elt-2 and rpt-6 were effectively inhibited in the elt-2(RNAi) and elt-2(RNAi);rpt-6(RNAi) animals. Next, we aimed to determine whether rpt-6 also played a role during recovery from bacterial infection. While RNAi downregulation of rpt-6 prevented the activation of two out of the four tested genes involved in recovery from P. aeruginosa infection (Fig 1E), rpt-6(RNAi) animals failed to recover from the infection (Fig 1F). These results suggest that rpt-6 and elt-2 may work together to control the expression of a subset of immune genes.