ATG9A localisation depends on AP-4 not only in HeLa cells, but also in neuroblastoma-derived SH-SY5Y cells and in fibroblasts from AP-4-deficient patients (Fig. 3 and Fig. 4), suggesting that trafficking of ATG9A from the TGN is a ubiquitous function of AP-4. Here, ATG9A is linked to neuroblastoma.