A recent study has demonstrated that lncRNA NUTF2P3-001 acts as a ceRNA to communicate with KRAS by competitively binding to hsa-mir-3923, and the up-regulation of NUTF2P3-001 reverses the suppressive effect of hsa-mir-3923 on KRAS, leading to the proliferation and invasion of pancreatic cancer [13]. The gene discussed is KRAS; the disease is pancreatic neoplasm.