PPARG and inflammatory bowel disease: Although the influence of Se and PPARγ on the evolution of IBD and the mechanism of Se impact on oxidative stress in this context have not represented the focus of many studies, to our knowledge, there has not been a research core on the effect of Se and PPARγ on IBD and, based on the available data, one could infer, using murine models of IBD [73, 74], that given the reduction in PPARγ in IBD, as well as the potential of Se to enhance PPARγ and its ligand 15d-PGJ2 [75], Se supplementation would significantly decrease disease activity.