SLC2A4 and type 2 diabetes mellitus: Concerning miR-199a, reported to be increased in plasma of T2DM subjects, it was described as capable of repressing GLUT4 (39) and HK2 (60), by means of gain- and loss-of-function approaches, in transfected HEK293T, L6, Huh-7 and/or HepG2 cells.