SOAT1 and infection: Expression of 010 reduced the activation of an IFN-β reporter upon infection with Sendai virus (SeV), a strong inducer of IFN-β production (Fig. 4B), but did not affect the activation of the antiviral signaling mediated by the Janus kinase/signal transducers and activators of transcription (JAK/STAT) pathway in response to IFN-β (Fig. 4C).