Others have found an interaction between smoking and maternal excision repair cross-complementation group 1 (ERCC1), ERCC5, poly(ADP-ribose)polymerase 2 (PARP2) genes and the infant o-sialoglycoprotein endopeptidase (OSGEP) gene, making the presence of these polymorphisms more likely to result in CHD when the mother smoked [25]. This evidence concerns the gene PARP2 and coronary artery disorder.