TOLLIP and idiopathic pulmonary fibrosis: Evidence is accumulating that chronic low‐level activation of the innate immune system has an important role in IPF, with several Tollip gene polymorphisms linked with the disease.31 A negative regulator of acute inflammation, Tollip also augments chronic low‐grade inflammation in a process that involves its translocation to mitochondria.32 Many of the inflammatory gene changes reported in this study align with our current understanding of IPF pathology and/or processes of cellular senescence.