REN and myocardial infarction: The transcription of cyto‐renin is under the control of an alternative promoter located in intron 1.7 This promoter has very low basal activity, but is stimulated by glucose depletion in cardiac cells7 as well as after myocardial infarction in the rat heart.8 Cells overexpressing cyto‐renin are more resistant against necrotic death under ischaemic conditions such as glucose depletion.9 Moreover, overexpression of cyto‐renin in the heart of transgenic rats reduces infarct size in ex vivo Langendorff preparations.9