The important role of EMT and its intermediate states have been extended to cancer cell migration and invasion.1 Cell plasticity owned in the process of EMT is associated with cancer stem cell‐like features and increased resistance to both radiotherapy and chemotherapy.2, 3, 4 During the EMT process, the adherens and tight junction proteins E‐cadherin and ZO1 lost their expression, accompanied with the increased expression of the mesenchymal proteins, such as N‐cadherin, fibronectin and vimentin. This evidence concerns the gene CDH2 and cancer.