CSF1 and neoplasm: Our findings in favor of this notion are as follows: (i) the impaired TAM recruitment and NCCIT xenograft tumor growth after the ectopic overexpression of miR-125b in tumor cells, (ii) the abolished effects of miR-125b on NCCIT xenograft tumor growth by the deletion of host macrophages, (iii) the direct regulation of tumor-derived chemokines (e.g., CSF1, CX3CL1) for host TAM recruitment by miR-125b in tumor cells, and (iv) a set of miRNAs under miR-125 regulation combinatorially target CSF1 and CX3CL1 in NCCIT cells.