In contrast, NAC treatment could block the growth and metastasis of MHCC97L cells induced by MTP18 overexpression, as evidenced by wound-healing and transwell invasion assays (Fig. 7c–f), indicating that increased mitochondrial fission and subsequent ROS production was involved in the oncogenic property of MTP18 in HCC. The gene discussed is MTFP1; the disease is hepatocellular carcinoma.