MiR-126 suppressed the growth of CRC neoplastic partly by targeting the p85β subunit of phosphatidylinositol 3-kinase (PI3K) which is highly correlated to cell survival and carcinogenesis through phosphorylation of downstream effectors, such as NF-κB and mechanistic target of rapamycin kinase (mTOR) [89]. This evidence concerns the gene MTOR and colorectal carcinoma.