For example, specific muscle-enriched miRNAs (the myomiRs; miR-1a-3p, miR-133a-3p, and miR-206-3p) regulate myoblast proliferation and differentiation during muscle growth and development.2, 3, 4, 5, 6 Furthermore, a set of miRNAs is differentially expressed in the muscles of Duchenne muscular dystrophy (DMD) patients and dystrophic animal models,7, 8, 9, 10 where they contribute to disease-associated processes such as muscle regeneration,11, 12 inflammation,13 fibrosis,14 and the regulation of dystrophin expression.15 The gene discussed is DMD; the disease is Duchenne muscular dystrophy.