This condition is accompanied by increased lipolysis and very-low-density lipoprotein secretion, [38] which leads to atherogenic dyslipidaemia (elevated low-density and decreased high-density lipoprotein concentrations), [39] hyperglycaemia due to impaired insulin sensitivity and glucose hyperproduction, and the increased release of inflammatory factors, such as IL-6, TNF-α, and C-reactive protein [40]. This evidence concerns the gene INS and Hyperglycemia.