However, it is worth noting that GWAS for dyslexia have been under‐powered so far and variants with the strongest association to dyslexia (e.g., in genes RBFOX2, ABCC13, ZNF385D, COL4A2 and FGF18) failed to survive genome‐wide statistical scrutiny (Eicher et al., 2013; Field et al., 2013; Gialluisi et al., 2014; Luciano et al., 2013). The gene discussed is ZNF385D; the disease is dyslexia.