As such, it is unlikely that risk variants in genes such as KIAA0319 or DCDC2 are sufficient to lead to defects in neuronal migration or other neurodevelopmental pathways contributing to dyslexia—particularly given some of these risk variants are also commonly found in non‐dyslexia populations—and thus must co‐occur with other factors. Here, KIAA0319 is linked to dyslexia.