Patients with the more recently described MOG antibodies typically associate with phenotypes of optic neuritis, longitudinally extensive myelitis, and acute disseminated encephalomyelitis.73 Although the latter often shows basal ganglia and thalamic imaging changes, there are only rare descriptions of movement disorders in patients with MOG antibodies in addition to the few with ataxia.74 This evidence concerns the gene MOG and optic neuritis.