Unlike the above-mentioned studies, this study additionally comprised in vitro experiments, which showed that in vitro siRNA-mediated knockdown of AGAP2-AS1 in GBM cell lines L215N229 and U87MG inhibited cell proliferation while the downregulation of AGAP2-AS1 suppressed invasiveness and increased the rate of apoptosis [67]. Here, AGAP2 is linked to glioblastoma.