For example, amplification of lncRNA SOX2‐OT was mutually exclusive with FOXA1 with contribution reaching 92.1% in LUSC (Fig. 3C), whereas amplification of lncRNA LINC‐PINT was mutually exclusive with EGFR, SGCB and CDKN2C in GBM with only 5.4% of contribution (Fig. 3D). The gene discussed is FOXA1; the disease is glioblastoma.