On the other hand, Cp not only provided nutrients for the gut microbiota, but also restored intestinal homeostasis in mice with colitis by upregulating beneficial bacteria in the gut and downregulating the abundance of harmful bacteria, and the recovered gut microbiota utilized metabolic enzymes encoded by its gut microbiome to further promote the decomposition of Cp into small molecule polysaccharides, ultimately promoted the production of SCFAs, and provided sufficient energy for the colon. This evidence concerns the gene CP and colitis.