Consistent with this high malaria attack rate is a high prevalence for RBC polymorphisms (Tables 1–3): 20% for HbAS, 14% for G6PD*A- (6% hemizygous males, 7% heterozygous females, 1% homozygous females), and an even higher prevalence of 3.7 α+-thal deletions in heterozygous (-α/αα, 46%) and homozygous (-α/-α, 10%) forms. Here, G6PD is linked to malaria.