G6PD and malaria: The high prevalence of RBC polymorphisms detected in this study (Tables 1–3): 20% for HbAS, 12% for G6PD*A-, and an even higher prevalence of 3.7 α+-thal deletions in heterozygous (-α/αα), 46%) or homozygous (-α/-α, 10%) forms are indicative of the strong selective pressure that malaria has exerted on these traits and are consistent with studies done in other malaria endemic regions in Sub-Saharan Africa [30–34].