Furthermore, some genes may be completely lacking in the zebrafish, such as the ENaC (epithelial Na channel) subunits (SCNN1A, SCNN1B, SCNN1G, and SCNN1D) [166], mutations in which cause Liddle syndrome and pseudohypoaldosteronism type 1 in humans. This evidence concerns the gene SCNN1G and pseudohypoaldosteronism type 1.