Chronic myeloid leukemia (CML) is a myeloproliferative neoplasm characterized by cytogenetic aberration, namely, the translocation of the Philadelphia chromosome (Ph) [1], which generates a breakpoint cluster region-Abelson murine leukemia viral oncogene homolog 1 (BCR-ABL1) fusion oncogene that encodes the BCR-ABL oncoprotein [2]. Here, ABL1 is linked to myeloproliferative neoplasm.