In order to model the environment generated by influenza infection of the lung, in a previous study we comprehensively compared the maturation profiles (phenotypic expression, cytokine production, apoptosis, and T cell proliferation) of untreated monocytes, IL-4-treated dendritic cells (DCs), IFN-treated DCs, and DCs following co-culture with supernatant from influenza infected lung epithelial cells. This evidence concerns the gene IFNA1 and influenza.