Individuals with mutations in the FOXP2 gene have problems executing coordinated sequences of orofacial movements, which impede their speech [diagnosed as developmental verbal dyspraxia or childhood apraxia of speech (CAS)], while their general cognitive functioning and other aspects of motor coordination are usually less severely affected (MacDermot et al. 2005; Morgan et al. 2017). This evidence concerns the gene FOXP2 and childhood apraxia of speech.