IL10 and infection: Specialized pro-resolving mediators may contribute to eradicate infections and terminate the inflammatory input, as it is suggested by murine sepsis models, where there was reported that LXA4 decreased plasma IL-6, chemokine (C–C motif) ligand 2 (CCL2), IL-10, and NF-κB activity in peritoneal macrophages, reduced neutrophil migration, and increased the clearance of bacteria by neutrophils without the production of excessive free radicals (Walker et al., 2011; Wu et al., 2015).