Particularly, both protein carbonyls, a marker of protein oxidation, and thiobarbituric acid reactive species (TBARS), a marker of lipid peroxidation, were increased along with IL-6 and IL-10 levels in patients with sepsis, both proven or clinically highly probable, in comparison to controls, and in patients with proven sepsis in comparison to controls [54]. The gene discussed is IL6; the disease is Sepsis.