Although PPARβ/δ was actually involved in regulation of PCa cell proliferation (Supplementary Figure 3B and 3C), FABP5 expression level had no impact on the phosphorylation level of AKT (Figure 3D), indicating that the FABP5-PPARβ/δ signaling axis might be physiologically inactive in PCa cells. Here, AKT1 is linked to posterior cortical atrophy.