To investigate the relationship between PPARβ/δ activity and FABP5 in PCa cells, we analyzed expression levels of PPARβ/δ target genes, 3-phosphoinositide-dependent protein kinase-1 (PDPK1), Adipose differentiation-related protein (ADRP) and Integrin-linked kinase (ILK), in FABP5 overexpressed PNT2 cells with/without highly selective PPARβ/δ agonist GW0742, which can also bind to FABP5 [32]. The gene discussed is FABP5; the disease is posterior cortical atrophy.